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Obesity results from the failure of the homeostatic mechanisms that normally regulate energy intake and expenditure An excess of intake over expenditure is stored as triglyceride, mainly in adipose tissue Food intake in adult humans is generally 2000 2500 kcal/day Human feeding is heavily influenced by psychological, social and economic factors Very rarely, human obesity is due to hyperphagia resulting from mutations affecting the regulatory circuits that normally restrain appetite (eg leptin, leptin receptor, cholecystokinin receptor) Obese people expend more energy than lean, and at stable weight, must therefore consume more energy to maintain their higher weight In general, obese subjects under-report their energy intake Overconsumption of energy-dense food and drinks is an important factor in the spread of obesity Energy as adenosine triphosphate (ATP) is generated by the oxidation of fats (yielding 9 kcal/g), carbohydrates and proteins (both yielding 4 kcal/g).

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s 25, 26: CAD 20: Database Knowledge Image Processing Data Mining 20: HIS/RIS/PACS Medical Images 22: ePR 20: Visualization Graphics Tool 22: ePR/Image 23: RT 24: MISS 20: Security Networks SAN Archive MIII 21: DATA GRID

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The respiratory quotient (RQ) the ratio of carbon dioxide produced to oxygen consumed during oxidation varies between 10 for carbohydrate and 07 for fat, and can be used to determine the principal substrate being utilized Total energy expenditure comprises resting energy expenditure (REE; about 60% of total), the thermic effect of food (ie the energy required for digestion and assimilation of ingested nutrients), and activity thermogenesis, which consists of both physical activity and non-exercise activity thermogenesis (NEAT) REE is determined mainly by fat-free mass, and 80% is due to the metabolic activity of the brain, heart, liver and kidneys; muscle contributes only 20% In an average adult, REE is about 13 kcal/min The contribution of physical activity is highly variable, accounting for less than 30% of total energy expenditure in sedentary subjects but 70% or more in highly-trained athletes or manual labourers.

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NEAT is involuntary, spontaneous physical activity, which varies widely between individuals Subjects with low levels of NEAT may be more susceptible to obesity, whereas increases in NEAT may help to limit weight gain during overeating Adaptive thermogenesis describes changes in energy expenditure that result from alterations in energy balance Variations in this could partly determine susceptibility to obesity; differences in sympathetic-mediated thermogenesis for example in muscle or functional brown adipose tissue (BAT) could contribute, but its significance remains uncertain Obesity could also develop through defects in fat oxidation, and there is evidence of decreased fat oxidation rates in some obese subjects (which can persist after weight loss) and in subjects who subsequently become obese..

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